EMF Connection?

Print Friendly, PDF & Email

This is not a comprehensive list, simply some articles I have found along the way.

Many children on the autism spectrum demonstrate drastic behavioral improvements by reducing exposure to electromagnetic frequency (EMF’s), this includes wifi/wireless routers, cellphones, smart meters, cellphone towers, etc.. This behavioral improvement may be because EMF’s increase overall glutamate levels, they cause a surge in glutamate from increased glial activity and they induce hyper stimulation of glutamate receptor (increasing sensitivity to glutamate). These high levels of glutamate also promote and contribute to mitochondria damage and decreases mitochondrial function. As we’ve previously discussed, excess glutamate is a primary mechanism behind mito dysfunction (often associated with low tone/motor delays, decreased motility, poor temperature regulation, increased hunger, delayed growth, etc.). I personally wonder if this is why many witness increased stimming, movements and tics with screen time??? If you didn’t have enough motivation to scale back on screen time and limiting EMF exposure, now you do.

“Given the ubiquity of the factors contributing to our glutamate load, and the pervasiveness of spectrum-wide disorder, a closer look at what conditions trigger glutamate receptor hyperactivity is imperative. It has been suggested that one of the first responses to overabundance of glutamate is creation of new receptors to compensate. High IQ has been correlated to greater numbers of glutamate receptors in the brain. In autism, genius is frequently observable in very restricted areas of ability, in conjunction with debilitating dysfunction from excitotoxicty. Perhaps with other pheno/endotypes, receptors multiply in differing areas of the CNS, or outside it. In addition to increase, glutamate receptors become hypersensitive. Exactly how and why has been perplexing. Ionotropic glutamate receptors are ligand gated ion channels. Quantum physics elucidates the loss of control over calcium voltage channels with the explanation of the biologic effect of non-native EMF. Due to the role of calcium ions, both NMDA and non-NMDA receptors are greatly influenced by non-native EMF.

In addition, non-NMDA receptors rely upon mitochondrial function, also affected by EMF. This impairs them by reduced ability to pump ions back out of the cell, resulting in increased sensitivity of NMDA receptors to glutamate molecules. Any increase in intracellular calcium is problematic in the presence of excess glutamate, as calcium is the agent that actually inflicts harm to the cell by activating a cascade of actions that cause cell death. Glutamate may be the gun, and calcium the bullet- but now we have an idea why the trigger is being continually fired. EMF creates a scenario where calcium efflux from surrounding tissues provides abundant intracellular free calcium ions.” “EMF links glutamate toxicity, mitochondrial dysfunction, and oxidative stress intrinsically through ionotropic glutamate receptor dysfunction- underpinning a host of neuroimmunological conditions.” https://forum.jackkruse.com/index.php?threads/emf-an-alternate-neurodiverse-theory-of-autism-and-glutamatergic-dysfunction.17182/

Radiofrequency increasing glutamate: Our results demonstrate that RF exposure enhanced glutamate-induced cytotoxicity by further increase of ROS production in HT22 cells. http://www.tandfonline.com/doi/abs/10.1080/09553002.2017.1237058?src=recsys&journalCode=irab20

EMF causing glial activity resulting in glutamate release http://www.sciencedirect.com/science/article/pii/S0969996104001573

“Our data demonstrate, for the first time, that even acute exposure to low intensity EMF induces ROS production and DNA fragmentation in astrocytes in primary cultures, which also represent the principal target of modulated EMF. Our findings also suggest the hypothesis that the effects could be due to hyperstimulation of the glutamate receptors, which play a crucial role in acute and chronic brain damage.” Reactive oxygen species levels and DNA fragmentation on astrocytes in primary culture after acute exposure to low intensity microwave electromagnetic field http://www.sciencedirect.com/science/article/pii/S030439401000176X

EMF and Mitochondrial Function

Effect of magnetic field on the process of cell respiration in mitochondria of rats https://www.ncbi.nlm.nih.gov/pubmed/3022316

 

EMF’s glutamate

Variations of glutamate concentration within synaptic cleft in the presence of electromagnetic fields https://www.ncbi.nlm.nih.gov/m/pubmed/25577979/

EMF’s increase glutamate- Extremely low frequency electromagnetic field exposure causes cognitive impairment associated with alteration of the glutamate level https://www.ncbi.nlm.nih.gov/pubmed/25066354