This is not a comprehensive list, simply some articles I have found along the way. There seems to be growing evidence that excess glutamate is also involved with dyslexia. " The first theory suggests that dyslexia might be a consequence of neuronal hyperexcitability, which contributes to learning deficits by heightened noise and instability in information processing3. This theory emphasizes the importance of balanced levels of excitation and inhibition within cortical pyramidal-interneuron networks, which support tuning to sensory input, neural timing, and information processing. Excitation–inhibition balance and the precise timing of neural activity can be disrupted by [...]